Bebidas edulcorantes y su riesgo para la salud
Abstract
Although the primary taste sensations are sweet, salty, sour and bitter, the vast majority of people choose foods and beverages with a great sweet taste. From a health point of view, one option to replace these beverages is non-caloric sweeteners which can help in the control of some chronic diseases since the Mexican population has a high consumption of caloric sweetened beverages such as soft drinks, juices and tea with added sugar and non-caloric sweeteners can easily replace the sweet taste of sugar. However, when consuming non-caloric beverages, a proper diet is recommended along with physical activity, because there is no scientific evidence that a "perfect" sweetener exists. There is currently information about the possible risks and benefits of the use of sugar-substitute sweeteners, however, more scientific research is needed to provide conclusive data about their effectiveness and long-term safety.
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References
[2] S. E. Swithers, A. A. Martin, y T. L. Davidson, High-intensity sweeteners and energy balance", Physiol. Behav., vol. 100, num. 1, pp. 55{62, abr. 2010.
[3] G. M. Singh et al., Estimated Global, Regional, and National Disease Burdens
Related to Sugar-Sweetened Beverage Consumption in 2010.", Circulation, vol. 132, num. 8, pp. 639-66, ago. 2015.
[4] S. J et al., Artificial Sweeteners Induce Glucose Intolerance by Altering the Gut Microbiota", Nature, vol. 514, n_um. 7521, 2014.
[5] J. M. Taylor, M. A. Weinberger, y L.Friedman, Chronic toxicity and carcinogenicity
to the urinary bladder of sodium saccharin in the in utero-exposed rat", Toxicol. Appl. Pharmacol., vol. 54, n_um. 1, pp. 57-75, jun. 1980.
[6] Y. Yu et al., \Risk factors for bladder cancer: A case-control study in Northeast
China", Eur. J. Cancer Prev., vol. 6, num. 4, pp. 363-369, 1997.
[7] G. D. Wu et al., \Linking long-term dietary patterns with gut microbial enterotypes",
Science (80-. )., vol. 334, num. 6052, pp. 105-108, oct. 2011.
[8] M. S. A. Palmnäs et al., Low-dose aspartame consumption diferentially affects gut microbiota-host metabolic interactions in the diet-induced obese rat", PLoS One, vol. 9, n_um. 10, oct. 2014.
[9] J. L. Kuk y R. E. Brown, Aspartame intake is associated with greater glucose intolerance in individuals with obesity", Appl. Physiol. Nutr. Metab., vol. 41, num. 7, pp. 795-798, mar. 2016.
[10] S. S. Gul et al., Inhibition of the gut enzyme intestinal alkaline phosphatase may explain how aspartame promotes glucose intolerance and obesity in mice", Appl. Physiol. Nutr. Metab., vol. 42, n_um. 1, pp. 77-83, 2017.
[11] G. S. Portela, R. Azoubel, y F. Batigalia, Effects of aspartame on maternalfetal and placental weights, length of umbilical cord and fetal liver: A kariometric experimental study", Int. J. Morphol., vol. 25, num. 3, pp. 549-554, 2007.
[12] C. E. Bryant, L. K. Wasse, N. Astbury, G. Nandra, y J. T. McLaughlin, Nonnutritive sweeteners: No class effect on the glycaemic or appetite responses to ingested glucose", Eur. J. Clin. Nutr., vol. 68, num. 5, pp. 629-631, 2014.
[13] Y. Liang, G. Steinbach, V. Maier, y E. F. Pfei_er, The effect of artiffcial sweetener on insulin secretion: 1. The effect of Acesulfame K on insulin secretion in the rat (studies in vivo)", Horm. Metab. Res., vol. 19, num. 6, pp. 233-238, 1987.
